The findings, published in Nature Immunology, indicate that tumors edit the activity of multiple genes to avoid immune ...

This important study using engineered mouse models provides a first and compelling demonstration of a pathogenic phenotype associated with lack of expression of p53AS, an isoform of the p53 protein ...

A team led by Sylvain Meloche, Professor in the Faculty of Medicine at UdeM and Director of IRIC's Signaling and Cell Growth ...

Mice treated with the gene therapy remained cancer-free for at least 100 days and resisted cancer resurgence in a lab test.